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Home » ‘Sailor’s disease’ scurvy linked to young boy’s death
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‘Sailor’s disease’ scurvy linked to young boy’s death

News RoomNews RoomSeptember 3, 2026No Comments
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‘Sailor’s disease’ scurvy linked to young boy’s death

Robyn Wuth

September 3, 2026 — 12:51pm

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A six-year-old Brisbane boy has died from scurvy, the so‑called “sailor’s disease” common in the convict era.

Connor Dean died after a cardiac arrest under general anaesthetic at Queensland Children’s Hospital in 2024.

The boy was admitted to Queensland Children’s Hospital in July, and died less than a month later.Brisbane Times

Two years later, Deputy State Coroner Stephanie Gallagher has released her findings, ruling the child’s fatal brain damage was most likely triggered by vitamin C deficiency and scurvy.

“Although the precise mechanism leading to the cardiac arrest could not be definitively determined, the treating clinicians and forensic pathologist considered vitamin C deficiency to be the most likely underlying factor,” she determined.

Issues with an unnamed multivitamin were also canvassed.

Scurvy was once common among sailors and convicts who went without fresh fruit and vegetables on long sea voyages.

However, it is now so rare in Australia that serious cases are almost always found and treated.

Connor had Down syndrome, was born with a serious heart defect that was repaired in 2018, and had delayed development, speaking mostly in single words and walking with a wide gait.

By July 2024, he had about three weeks of worsening leg pain.

He was admitted to hospital on July 18, with X‑rays and MRIs failing to show a clear cause.

He then developed a bad cough and tested positive for parainfluenza as his oxygen levels and kidney function worsened.

Scans of his skeleton later showed bone changes “in keeping with scurvy”.

Blood tests revealed a vitamin C level of less than one micromole a litre, compared with a normal range of 20 to 120 micromoles a litre.

He was also low in vitamins A and E, had borderline low vitamin D and blood‑clotting problems that suggested vitamin K deficiency.

Connor never recovered from the brain damage after the cardiac arrest and died on August 10.

His paediatrician told the court he and dietitians had long‑standing concerns about the boy’s growth, with his height and weight well below the usual range even for children with Down syndrome.

“He was reported to have become very fussy with his intake and his diet was said to contain no fruit or vegetables at that time,” the paediatrician said.

“He had only gained 300 grams in the last 11 months.”

Connor was prescribed multivitamins.

However, during his final admission, doctors found the multivitamin the family was using, known in the findings only as “the Product”, did not contain vitamin C.

“I am concerned that a product such as [the one Connor was taking] is available in Australia. I believe that it is misleading to call this product a multivitamin when it misses essential vitamins such as vitamin C,” the pediatrician said.

“It is my clinical opinion that Connor would not have had vitamin C deficiency, and the clinical features of scurvy that led to his death, if he had been taking a different multivitamin that contained vitamin C.”

The multivitamin’s manufacturer offered “sincere condolences” but rejected the paediatrician’s criticism and said there was “no basis on which to draw the conclusion that [its multivitamin] has contributed to Connor’s death”.

The coroner agreed there was not enough evidence to say the multivitamin caused or contributed to the death.

Instead, she focused on the failure to detect Connor’s severe vitamin C deficiency in time.

AAP

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